Time of Our Lives: The Science of Human AgingOxford University Press, 2001. 1. 11. - 288ÆäÀÌÁö By the year 2050 one in five of the world's population will be 65 or older, a fact which presages profound medical, biological, philosophical, and political changes in the coming century. In Time of Our Lives, Tom Kirkwood draws on more than twenty years of research to make sense of the evolution of aging, to explain how aging occurs, and to answer fundamental questions like why women live longer than men. He shows that we age because our genes, evolving at a time when life was "nasty, brutish, and short," placed little priority on the long-term maintenance of our bodies. With such knowledge, along with new insights from genome research, we can devise ways to target the root causes of aging and of age-related diseases such as Alzheimer's and osteoporosis. He even considers the possibility that human beings will someday have greatly extended life spans or even be free from senescence altogether. Beautifully written by one of the world's pioneering researchers into the science of aging, Time of Our Lives is a clear, original and, above all, inspiring investigation of a process all of us experience but few of us understand. |
¸ñÂ÷
1 | |
2 Attitudes to ageing | 12 |
3 Whats in a name? | 22 |
4 Longevity records | 39 |
5 The unnecessary nature of ageing | 52 |
6 Why ageing occurs | 63 |
7 Cells in crisis | 81 |
8 Molecules and mistakes | 100 |
12 Eat less live longer | 174 |
13 Why do women live longer than men? | 184 |
14 The Genie of the Genome | 196 |
15 In search of WonkaVite | 212 |
16 Making more time | 230 |
Epilogue | 243 |
Notes | 257 |
Bibliography | 261 |
±âŸ ÃâÆǺ» - ¸ðµÎ º¸±â
ÀÚÁÖ ³ª¿À´Â ´Ü¾î ¹× ±¸¹®
ageing process allele Alzheimer's disease amino acid animals antioxidant apoptosis become biological blood body bone brain called Calment cancer cause cell division cellular centenarians chemical child chromosomes copies culture damage death gene developed dietary restriction disposable soma theory effects enzyme evidence evolutionary evolved female fraitch free radicals genetic germ cells germ-line grow growth happen Hayflick Limit Hoddle hormone human hydra idea immune system important increase individual kinds live longer look maintenance male mechanisms memory menopause mice Miranda mitochondria mobbit molecular molecules mortality mutations natural selection Navrongo neurones normal occurs old age older oncogenes organs parents population problem proteins repair replicative reproduction result ribosome saw in Chapter scientific semelparous soma somatic cells span species sperm stem cells straws suggested survival syndrome telomerase telomeres things tion tissues tRNA tumour Werner's syndrome women Wonka-Vite young